5-aminolevulinic acid is an endogenous precursor of protoporphyrin IX, which is a photosensitiser. The medication ALA is used topically in conjunction with blue light illumination for the treatment of some grades of actinic keratoses of the face and scalp. ALA solution is applied topically. It is not intended for application by patients or unqualified practitioners. Application should involve either scalp or face lesions, but not both simultaneously. Contact your cosmetic dermatologist for more information.


5-ALA 5-Amino Levulenic Acid
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Bleach and Fade Gel
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Clobetasol Cream
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Collagen Cream
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Estradiol Cream
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Estriol Cream
  • Prescription Only


The coincidence of climacteric symptoms and the beginning of skin aging is directly associated with estrogen deficiency. As we age into menopause the estrogen levels begin to fall with the subsequent aging and lining of the skin. By application of refined estrogens (either estriol perimenopausally or estradiol menopausally) this process may be reversed by use of this therapy.

Available on prescription only


A dermatological cream has three primary functions :

  • To protect the skin from the environment and permit skin rejuvenation
  • To provide for skin an emollient or hydration effect
  • To provide a means of conveying medication to the skin for a specific effect either systemically or, as in this case, locally.

The cream is elegant, nonallergic, nonsensetising, low-irritant and stable.


The coincidence of climacteric symptoms and the beginning of skin ageing suggests that estrogen deficiency may be a common and important factor in both the peri- and menopausal woman. Often hormones have been considered important in endogenous ageing of skin. Thus by using topical application of estrogens this ageing may be reversed eg by either estradiol 0.01% cream for the menopausal and postmenopausal woman, or estriol 0.3% cream for the premenopausal woman- available only on prescription. From research we have found that elasticity and firmness was markedly improved after a period of six months, and wrinkle depth and pore size had decreased by 61-100%. Furthermore, skin moisture, type III collagen and the number of collagen fibres all improved dramatically. With all these external benefits no systemic absorption was found, and thus no estrogenic side effects occurred. From studies done it has been found that at the and of a six month clinical trial, marked improvement of skin ageing symptoms was noted;

  • Clinical improvement of specific skin parameters was evaluated and was seen in 9-19 weeks with estradiol and 7-17 weeks with estriol.
  • Improvement in skin elasticity and firmness was noted after 13 weeks with estradiol and 11 weeks with estriol.
  • Improvement in skin moisture was noted after 9 weeks with estradiol and 8 weeks with estriol.
  • Improvement in wrinkle depth was noted after 16 weeks with estradiol and 17 weeks with estriol.
  • Reduction of pore size was noted after 19 weeks with estradiol and 16 weeks with estriol.

All the above improvements appeared in 61–100% of cases.

In more detail wrinkle depth reduction was significant with estradiol and highly significant with estriol. Side effects were more pronounced with the estradiol group than the estriol group.

Both estradiol and estriol exhibited significant effect on increasing collagen fibre and striking increases in collagen III. This is why skin was firmed and wrinkles reduced. Type I collagen is predominantly in adult skin while type III is although distributed about the body is predominant in fetuses.

The positive effect of the estrogens on hydration was noted in all patients. This may be due to the increase in skin thickness with subsequently elevated amounts of natural moisturising factor.


By considering the results of topical estrogen treatment in skin ageing in women, a better insight can be gained as to the hormonal aspects of endogenous ageing of the skin. Various structures involved in skin ageing are under hormonal control. If decreased estrogen levels contribute to decreased functions of the skin, local estrogen treatment of the skin would in turn represent a local hormone substitution therapy of the skin. So far, estrogen compounds and, in particular estriol represent a new and promising therapeutic approach towards skin ageing in peri and menopausal women.


5-ALA 5-Amino Levulenic Acid
  • Prescription Only
Alpha Lipoic Acid
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Chromium Picolinate
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Co Enzyme Q10 capsules
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Estradiol Injections
  • Prescription Only
Ferronyl/Carbonyl Capsules
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Lactase Capsules
  • Order
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Max B Forte
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Manganese Chloride solution
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Migraine Nasal Spray
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Nicotinic Acid, Niacin Capsules
  • Order
  • Prescription Only
Probiotic Pessaries
  • Order
Selenium capsules
  • Prescription Only
Thyroid Extract
  • Prescription Only
Tri-iodothyronine Capsules
  • Prescription Only
Vitamin B12
  • Order
Vitamin D3 Cholecalciferol
  • Prescription Only


Arthritis is a degenerative joint disease that causes pain, inflammation and limited joint movement.

In osteoarthritis the joint that is affected has degenerated cartilage. As this is the cushion between the joints, one gets bone rubbing against bone and thus pain, inflammation and lack of mobility follows. Standard treatment with nonsteroidal anti-inflammatory drugs (e.g. Brufen, Orudis) or by injection with steroids (e.g. cortisone). mask the symptoms and relieve the pain as the disease progresses and deleterious side effects may occur.

Since the mid 1990s, excellent results have however best achieved with a combination of natural nutritional supplementscombining glucosamine and chondroitin sulphate.

Firstly healthy cartilage needs three things: water for lubrication and nourishment, proteoglycans to attract and hold the water, and collagen to keep the proteoglycans in place.

Proteoglycans are like a rope that threads itself through the collage and are essential as they hold many times their own weight of water, which both lubricates and nourishes the collagen. If the cartilage is damaged the thread of rope becomes weak and ‘leaks’ out and thus the collagen loses its nourishment as the proteoglycans lose their grip and float away. Thus the cartilage cannot withstand shocks, cracks and may wear out completely.

Glucosamine is a major building block of the water- loving proteoglycans. In addition its presence acts as a stimulus to the cells that produce proteoglycans – in fact glucosamine is a key factor in determining how many proteoglycans are produced by the cells. Glucosamine has been shown to speed up production of both proteoglycans and collagen and it normalises cartilage metabolism which helps keep cartilage from breaking down.

Thus because of the affect of glucosamine on cartilage metabolism it can help the body to repair damaged or eroded cartilage.

Besides stimulating cartilage production, glucosamine also reduces joint pain and inflammation.

Where glucosamine helps form the proteoglycans that sit within the space in the cartilage, chondroitin sulphate acts like ‘liquid magnets’. Chondroitin is a long chain of repeating nigans that attracts fluid into the proteoglycan molecules. This is important for two reasons:

  1. the fluid acts as a spongy shock absorber
  2. the fluid sweeps the nutrients into the cartilage. Joint cartilage has no blood supply thus all of its nourishment and lubrication comes from the fluid that ebbs and flows as pressure is applied and released to the joint. Without this fluid, cartilage would become malnourished, drier, thinner and more fragile.


Chrondoiton Sulphate is a long chain molecule with a negative charge attached to it. As these chains wrap around proteoglycans they repel each other and thus create spaces between each proteoglycan. These are what are known as matrixes within the cartilage and this is where the fluid flows. There may be as many as 10,000 of these chains on a single proteoglycan molecule – thus we have a super water retainer as these chains make sure all these molecules are away from each other and cannot clump together.

Besides drawing in precious fluid, chondroitin :

  • protects cartilage and stops it from breaking down and inhibits certain ‘cartilage chewing’ enzymes
  • interferes with the metabolism of other enzymes that will starve the cartilage of fluid
  • stimulates production of proteoglycans, glucosamine and collagen.


Natural (Bio-identical) Hormones


7-keto DHEA, 7-keto dehydroepiandrosterone
  • Prescription Only
  • Prescription Only
DAP Caps
  • Prescription Only
DHEA tablets, troches, creams
  • Prescription Only
Estradiol Cream
  • Prescription Only
Estradiol Injections
  • Prescription Only
Estriol Cream
  • Prescription Only
Natural Progesterone Cream
  • Prescription Only
Testosterone and DHEA Troches
  • Prescription Only
Thyroid Extract
  • Prescription Only
Thyroxine T4 capsules
  • Prescription Only
Tri-iodothyronine Capsules
  • Prescription Only
Tri-iodothyronine, T3 capsules
  • Prescription Only
  • Prescription Only


Andropause (Male menopause) is caused by a decline in the hormone testosterone resulting in symptoms such as:

  • Decreased energy and strength
  • Increased body fat
  • Osteoporosis
  • Depression
  • Decreased mental acuity
  • Inability to maintain muscle
  • Loss of eagerness and enthusiasm
  • Increased risk of cancer
  • Increased risk of heart disease
  • Increased risk of arteriosclerosis
  • Decrease libido
  • Decreased sensitivity to stimulation
  • Decreased strength of orgasm
  • Decreased erectile function

Men often rationalise the symptoms as due to aging, stress and the pressure in mid-life.

Testosterone deficiency is easily diagnosed through laboratory tests and resolved through natural hormone replacement of testosterone and DHEA. The tests cover total testosterone, free testosterone and DHEA-Sulfate. It is also good to have reference levels of PSA, Estradiol and cholesterol.

Diseases of the blood vessels are one of the main causes of premature death in Australia. Conventional medicine has not yet accepted that male menopause or more particularly decreased androgen levels increase the risk atherosclerosis. There is evidence dating back half a century that:

  • Testosterone is a primary factor in the health of the heart and blood vessels
  • Testosterone levels decline with age
  • Restoring testosterone (and DHEA) to youthful levels can yield significant health benefits, including protection against various manifestations of arterosclerotic disease


Looking at the normal high fat diet eaten by most males one can see that this type of diet leads to the clogging of the arteries. If, and more likely when, these arteries go into spasm an occlusion follows and a stroke or heart attack follows. To remedy this situation one must look at dietary considerations and the taking of anti-oxidants as blood vessels with high levels of anti-oxidants have some protection.

Consider cholesterol – the LDL cholesterol protein carrier is more prone to oxidation. When this happens the body sees this as foreign and the immune system sends out macrophages that engulf the oxidised LDL in a foam cell that is then deposited on the arteries to form plaque that in turn clogs up the arteries.

Antioxidants reduce the likelihood of this chain reaction. Vitamin E is one of the best anti-oxidants for cardio-vascular tissue. There have been several studies that show that by taking vitamin E the risk of heart attack drops by 40%, and it will also help to remove plaque from artery walls.


Testosterone is produced in the testes and when too much testosterone is produced there is a feedback loop to the hypothalamus to make excess testosterone into estradiol. This estradiol then feeds back to the pituitary gland that then indirectly makes the hypothalamus shut down any more production of testosterone.

Other extraneous factors also influence the testosterone levels. If we eat too much meat that contains estrogens, the feedback is started and testosterone production is shut down – if we drink too much alcohol the loop is also activated, as alcohol is an estrogen producer. Fat cells also produce estrogens, so it can be seen that lots of us have too much estrogen and not enough testosterone.

There may be a biological point of no return when the normal balance of testosterone and estradiol starts tilting towards estradiol. When this happens the equilibrium between the two is put out of balance and estradiol seems to dominate. It is important that it is not the absolute value of each hormone but more likely the ratio between testosterone and estradiol. It has been found by various researchers that increased estradiol is directly linked with myocardial infarction – the opposite to women.


Although focusing primarily on testosterone, mention should be made of the beneficial effects of DHEA (dehydroepiandrosterone). Produced in the adrenal glands, DHEA lies at the heart of the steroid family tree. DHEA is descended from cholesterol by way of pregnenolone and is a direct precursor of androstenedione and androstenediol both of which are directly metabolised to testosterone.

DHEA normally reaches peak levels in the early twenties and then declines gradually so that by age eighty we have about 5% left. It has beneficial testosterone-like effects such as:

  • Feeling of energy and well being
  • Improved insulin sensitivity and glucose tolerance
  • Reduced death from coronary heart disease
  • Lower obesity and waist to hip ratio
  • Slowed progression of atherosclerosis
  • Enhanced libido and erectile ability
  • Reduced depression and enhanced cognition

Looking at the steroid family tree – we can see that cholesterol is at the top and it breaks down through various pathways to estradiol, testosterone and many other molecules. By simply adding a precursor we could assume that it slots itself in and things go on further down the pathway – for instance we take some DHEA and the testosterone level goes up accordingly. It’s not so simple, as sometimes this will work in some individuals and not in others. Hence constant monitoring of the situation must be performed to see if what we want to occur is actually occurring.


Once the testosterone levels drop so does general well-being.

  • Decreased libido and potency.
  • Early senility – as testosterone loss leads to brain cell memory failure.
  • Reduced mental agility.
  • Loss of ability to concentrate.
  • Moodiness and emotionality – grumpy old man syndrome caused by the imbalance in the testosterone/estradiol ratio.
  • Depression – this is a major problem for men who don’t talk about their problems.
  • Reduced activity in general – the couch potato syndrome. As the testosterone goes down you feel less energetic so you sit on the couch. This leads to putting on weight. Fat makes more estrogen. The estrogen tells the brain that you’ve got enough testosterone
    (remember the loop) leading to a downward spiral.
  • Less aggression, more passive – again think of the loop.
  • Nervousness, general tiredness, feeling weak, no oomph, anxiety etc.- these are all due to the estrogen/testosterone
    ratio being out of balance.


Vascular disease stroke or heart attack which is a major medical condition in Australian men and women. Testosterone addresses vascular disease in a number of ways.

Low testosterone affects us in the following ways:

  • Angina
  • Atherosclerosis
  • High blood cholesterol
  • High blood triglycerides
  • High blood pressure. High body mass index (obesity).

Testosterone has a major effect on changing the way your body processes insulin. High insulin levels lead to vascular disease, as it causes an increase in triglycerides, a decrease in HDL cholesterol that leads to clogging of arteries etc. Testosterone lowers LDL, raises UDL, reduces triglycerides (because of its effect on insulin)., reduces blood pressure, and changes the body mass index as it converts fat to muscle. It is worth noting that the two sexes tend to look alike as we get older and that’s because of the relative increase in estrogen.

It was, in fact found in a post-war study by Danish physician Jans Moller, that cholesterol levels dropped 83% in a study of 300 men. The average drop was 26%.

Dr. Moller was decades ahead of his time and tried to treat the cause of cardiovascular disease rather than its symptoms. Then, as now, the symptoms are, high cholesterol, hypertension, atherosclerosis, thrombosis, intermittent claudication, angina and other manifestations. The standard treatment has been to lower cholesterol, reduce blood pressure, and dilate coronary arteries (as well as replacing clogged arteries surgically as if they were clogged pipes). “This theory does not have anything to do with prevention and treatment of cardiovascular disease” wrote Moller.

Moller basically believed that the normal condition of living organism is a balance between anabolic (protein building) and catabolic (protein destroying) processes. The primary anabolic hormone is testosterone and the primary catabolic hormone is cortisol. Cardiovascular disease results when catabolic influences come to predominate, leading to an excess of cholesterol, impaired carbohydrate metabolism, decreased fibrinolysis and other symptoms.

Lowering cholesterol by reducing dietary intake and by use of drugs, can lead to impotence and impaired cardiac function. This is logical when one remembers that testosterone is made from cholesterol and testosterone improves libido and protects the heart.


There are two components to developing an erection. Firstly thoughts stimulate the production of testosterone, which in turn stimulates blood vessels in the penis to fill and thus form an erection. To maintain the erection the testosterone level goes down which stimulates the production of nitrous oxide which develops a couple of other products that dilate the vessels.

So to sustain an erection, simple testosterone supplementation will not work unless the blood vessels are free and clear to dilate effectively. This can be one of the first signs of coronary artery disease as this starts before the angina.

Thus as we get older, the gap between libido and potency gets wider.


Testosterone is found in the bloodstream in two forms, namely as free testosterone which is active, and the form bound to a steroid binding globulin, SHBG. As we get older the bound version rises but the free version falls. Blood results of total testosterone may show a small drop, when in fact from about 40 years of age free testosterone levels drop a lot, hence test both. Note that as the estrogen levels go up relatively so too does the SHBG, thus less free testosterone.

Testosterone supplementation increases the level of both total and free testosterone, and over a 12 month period the symptoms are reversed, the risk of coronary artery occlusion goes down, angina episodes go down, as do the other symptoms of testosterone deficiency.

Di-indolylmethane is an extract found in cruciferous vegetable s that has a number of actions:

  1. it increases the portion of testosterone that is free in serum (by altering the free: bound ratio)
  2. it alters the metabolism of estrogens in favour of the safer estrogens
  3. it increase the progesterone binding


On a daily basis men get a pulse of testosterone about midday, another about 3pm, and a big pulse between 3-8am. Thus the best way to mimic this is by using troches, taking them three times a day, or at least twice a day. Since the major secretion is in the morning and to improve compliance a single morning dose has proved to be adequate.

The actual dosage with troches varies, between 10-50mg twice daily, with age basically dictating the dose (i.e. the older the higher the dose). Sometimes doses as high as 200mg a day are necessary for such problems as vascular occlusion but this is rare. If there is an artery problem, often the last symptom to be corrected is erectile function, as that needs to be corrected before an erection can be maintained.


A large proportion of our community don’t receive enough exposure to sunshine to sustain recommended levels of Vitamin D in the body, especially during the winter months. Oral supplementation with higher-dose capsules is common place now, and there is a huge body of evidence showing the many health benefits of this vitamin.

Vitamin D is a steroid hormone that assists with calcium homeostasis and absorption in the gut, assists with the conversion of T4 to T3, helps the pancreas release insulin and is even necessary for blood clotting.

The 25-hydroxy D is the form that is tested by pathology labs – with a normal range of 50 to 140 nmol/L. Aiming for levels in the top one-third of this normal range is ideal.

A prescription is required for this medication for doses over 1,000IU.

Contact your chemist for more information.